What Is Progesterone?

Bioidentical Micronized Progesterone · Last reviewed by the OPTML clinical team on April 29, 2026

Quick answer

Progesterone is the principal hormone of the luteal phase of the menstrual cycle, produced by the corpus luteum after ovulation. Micronized progesterone (Prometrium, or compounded equivalent) is bioidentical, structurally identical to endogenous ovarian progesterone, and is the form used in modern HRT. It is distinct from synthetic progestins like medroxyprogesterone acetate (Provera, used in the original WHI trial), which are not bioidentical and have a different side-effect and risk profile.

Physiology

Progesterone is produced primarily by the corpus luteum following ovulation, with smaller contributions from the adrenal cortex and (during pregnancy) the placenta. Levels rise sharply in the luteal phase (typically 5-25 ng/mL) and fall to baseline if pregnancy does not occur. Progesterone declines progressively during perimenopause as ovulation becomes irregular, and reaches very low post-menopausal levels.

Progesterone acts on progesterone receptors (PR-A, PR-B) found in the uterus, breast, brain, and other tissues. Centrally, its allopregnanolone metabolite is a positive allosteric modulator of GABAA receptors, explaining the well-documented sedative and anxiolytic effects of oral micronized progesterone.

Forms

FormRouteNotes
Micronized progesterone (Prometrium)Oral capsule (nightly)Bioidentical; sedating effect; first-pass metabolism
Compounded micronized progesteroneOral or vaginalBioidentical; allows custom dosing
Vaginal progesteroneVaginal gel or insertHigher uterine concentrations; less sedation
Medroxyprogesterone acetate (Provera)OralSynthetic progestin; not bioidentical
Norethindrone, levonorgestrel, drospirenoneOralSynthetic progestins used in birth control / some HRT

Indications in HRT

Bioidentical Progesterone vs. Synthetic Progestins

The original 2002 WHI trial used medroxyprogesterone acetate (Provera), a synthetic progestin, not bioidentical micronized progesterone. The E3N observational cohort (Fournier et al., Breast Cancer Res Treat, 2008) found that the breast cancer risk associated with HRT was substantially lower when bioidentical micronized progesterone was used compared to synthetic progestins. This distinction is important: most modern HRT prescribing favors bioidentical preparations, and risk-benefit conversations should reference data on the specific formulation rather than treating "progesterone" and "progestins" as interchangeable.

Side Effects and Risks

Typical Lab Monitoring

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References

  1. Fournier A, Berrino F, Clavel-Chapelon F. Unequal risks for breast cancer associated with different hormone replacement therapies: results from the E3N cohort study. Breast Cancer Res Treat. 2008;107(1):103-111.
  2. Schumacher M, Mattern C, Ghoumari A, et al. Revisiting the roles of progesterone and allopregnanolone in the nervous system. Prog Neurobiol. 2014;113:6-39.
  3. The 2022 Hormone Therapy Position Statement of The North American Menopause Society. Menopause. 2022;29(7):767-794.
  4. Stuenkel CA, Davis SR, Gompel A, et al. Treatment of Symptoms of the Menopause: An Endocrine Society Clinical Practice Guideline. J Clin Endocrinol Metab. 2015;100(11):3975-4011.